Google Scholar: https://scholar.google.com/citations?user=TepFHoUAAAAJ&hl=en
Engi SA, Beebe EJ, Ayvazian VM, Cruz FC, Cheer JF, Wenzel JM*, Zlebnik NE* (2021). Cocaine- induced increases in motivation require 2-arachidonoylglycerol mobilization and CB1 receptor activation in the ventral tegmental area. Neuropharmacology 193: 108625.
Wenzel JM, Oleson EB, Gove WN, Cole AB, Gyawali U, Dantrassy HM, Bluett RJ, Dryanovski DI, Stuber GD, Deisseroth K, et al. (2018). Phasic Dopamine Signals in the Nucleus Accumbens that Cause Active Avoidance Require Endocannabinoid Mobilization in the Midbrain. Current Biology, 28(9):1392-1404.
Wenzel JM, Cheer JF (2018). Endocannabinoid regulation of reward and reinforcement through interaction with dopamine and endogenous opioid signaling. Neuropsychopharmacology, 43(1): 103-115.
Wenzel JM, Rauscher NA, Cheer JF, Oleson EB (2015). A role for phasic dopamine release within the nucleus accumbens in encoding aversion: A review of the neurochemical literature. ACS Chemical Neuroscience, 6(1): 16-26.
Ettenberg A, Cotten SW, Brito MA, Klein AK, Ohana TA, Margolin B, Wei A, Wenzel JM (2015). CRF antagonism within the ventral tegmental area but not the extended amygdala attenuates the anxiogenic effects of cocaine in rats. Pharmacology, Biochemistry and Behavior, 138:148-55.
Ettenberg A, Fomenko V, Kaganovsky K, Shelton K, Wenzel JM (2015). On the positive and negative responses to cocaine and their relation to drug self-administration in rats. Psychopharmacology, 232(13): 2363-75.
Covey DP, Wenzel JM, Cheer JF (2014). Cannabinoid modulation of drug reward and the implications of marijuana legalization. Brain Research, 1628(0): 233–243.
Wenzel JM, Cheer JF (2014). Endocannabinoid-dependent modulation of phasic dopamine signaling encodes external and internal reward-predictive cues. Frontiers in Psychiatry, 1(5): 118.
Wenzel JM, Cotton, SC, Dominguez HM, Lane JE, Shelton, K, Su Z-I, Ettenberg A (2013). Noradrenergic β-receptor antagonism within the central nucleus of the amygdala or bed nucleus of the stria terminalis attenuates the negative/anxiogenic effects of cocaine. Journal of Neuroscience, 34(10): 3467-74.
Wenzel JM, Su Z-I, Shelton K, Dominguez HM, von Furstenberg VA, Ettenberg A (2013). The dopamine antagonist cis-flupenthixol blocks the expression of the conditioned positive but not the negative effects of cocaine in rats. Pharmacology, Biochemistry and Behavior, 114-115; 90-96.
Su Z-I, Santoostaroam A, Wenzel JM, Ettenberg A (2013). On the persistence of cocaine-induced place preference and aversion in rats. Psychopharmacology, 229(1): 115-23.
Su Z-I, Wenzel JM, Ettenberg A, Ben-Shahar O (2013). Prior extended daily access to cocaine elevates the reward threshold in a conditioned place preference test. Addiction Biology, 19(5): 826-31.
Su Z-I, Kichaev G, Wenzel JM, Ben-Shahar O, Ettenberg A (2012). Weakening of negative relative to positive associations with cocaine-paired cues contributes to cue-induced responding after drug removal. Pharmacology, Biochemistry and Behavior, 100: 458–63.
Wenzel JM, Waldroup SA, Haber ZM, Su Z-I, Ben-Shahar O, Ettenberg A (2011). Effects of lidocaine-induced inactivation of the bed nucleus of the stria terminalis, the central or the basolateral nucleus of the amygdala on the opponent-process actions of self-administered cocaine in rats. Psychopharmacology, 217(2):221-30.
Su Z-I, Wenzel JM, Baird R, Ettenberg A (2011). Comparison of self-administration behavior and responsiveness to drug-paired cues in rats running an alley for intravenous heroin and cocaine. Psychopharmacology, 214(3):769-78.
Thiel KJ, Wenzel JM, Pentkowski NS, Hobbs RJ, Alleweireldt AT, Neisewander JL (2010). Stimulation of dopamine D2/D3 but not D1 receptors in the central amygdala decreases cocaine-seeking behavior. Behavioural Brain Research, 214(2): 386-94.